In studies of other receptor tyrosine kinases implicated in the oncogenesis of GIST, nilotinib realized strong and selective inhibition of PDGFRα and PDGFRβ. As is the case with imatinib, nilotinib potently inhibited the autophosphorylation of A31 cells transformed by PDGFRARestrictions of use: Not indicated for suppression of benign thyroid nodu

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